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Liver inflammation was evaluated by the expression of pro-inflammatory cytokines, and neutrophil infiltration

Varfolomeev, E

Our group, working with partially deficient IGF-1 mice, has demonstrated that the liver is capable of expressing IGF-1R (which under physiological conditions it does not [99]) as a defence mechanism (peer review)

Ferroptosisregulated by iron metabolism and lipid peroxidation (222)plays a non-redundant role in SLE/LN via multiple immune cell types: T/B-cell iron overload triggers ferroptosis to amplify autoimmunity (215, 218)

Neurons transfected with mRFP- -actin and stimulated with HGF, Dihexa or Nl l-AnglV significantly increased spinogenesis (* P 0.05